Whether cells in the human body survive or die under stress depends, among other things, on their mitochondria. Scientists at ...

Bax monomers then form dimer units, self-assemble into oligomers and generate pores in the MOM, leading to the caspase cascade by the release of cytochrome C. Figure 5. Bcl-2 and Bax regulate both ...

New research shows how glucose restriction in tumors protects cancer cells from chemotherapy by preserving pyrimidines and preventing apoptosis, offering insights into treatment strategies.

Whether cells in the human body survive or die under stress depends, among other things, on their mitochondria.

If the ATP in the mitochondria drops sharply, a protein important for apoptosis, known as cytochrome c, remains trapped in the mitochondria and the cell does not die, even if it receives the ...

The universal “fuel source” for cellular activity is ATP (adenosine triphosphate). If the ATP in the mitochondria drops sharply, a protein important for apoptosis, known as cytochrome c, remains ...

Correspondence to: Dr G J Gores Mayo Clinic College of Medicine, 200 First St SW, Rochester, Minnesota 55905, USA; gores.gregorymayo.edu Apoptosis is a highly organised and genetically controlled type ...

Bcl-2 is an apoptosis suppressor factor that regulates cell survival by controlling mitochondrial membrane permeability. It suppresses apoptosis by inhibiting the release of cytochrome C from ...

State Key Laboratory of Physical Chemistry of Solid Surfaces and Fujian Provincial Key Laboratory of Theoretical and Computational Chemistry, College of Chemistry and Chemical Engineering, Xiamen ...

The review also explores the anti-cancer effects of plant-mediated AuNPs, such as their ability to selectively target cancer cells and induce apoptosis, supported by both in vitro and in vivo studies.